Journal of International Oncology››2017,Vol. 44››Issue (7): 519-521.doi:10.3760/cma.j.issn.1673-422X.2017.07.010
Previous ArticlesNext Articles
Zhang Baihong, Yue Hongyun
Received:
2017-01-03Online:
2017-07-08Published:
2017-06-20Contact:
Zhang Baihong E-mail:bhzhang1999@126.comSupported by:
Natural Science Foundation of Gansu Province of China (1308RJZA181)
Zhang Baihong, Yue Hongyun. Cancer and aging[J]. Journal of International Oncology, 2017, 44(7): 519-521.
[1] Alexandrov LB, Jones PH, Wedge DC, et al. Clocklike mutational processes in human somatic cells[J]. Nat Genet, 2015, 47(12): 1402-1407. DOI: 10.1038/ng.3441. [2] Martincorena I, Campbell PJ. Somatic mutation in cancer and normal cells[J]. Science, 2015, 349(6255): 1483-1489. DOI: 10.1126/science.aab4082. [3] Smetana K Jr, Lacina L, Szabo P, et al. Ageing as an important risk factor for cancer[J]. Anticancer Res, 2016, 36(10): 5009-5017. DOI: 10.21873/anticanres.11069. [4] LópezOtín C, Blasco MA, Partridge L, et al. The hallmarks of aging[J]. Cell, 2013, 153(6): 1194-1217. DOI: 10.1016/j.cell.2013.05.039. [5] Finkel T, Serrano M, Blasco MA. The common biology of cancer and ageing[J]. Nature, 2007, 448(7155): 767-774. DOI: 10.1038/nature05985. [6] Ruhland MK, Coussens LM, Stewart SA. Senescence and cancer: an evolving inflammatory paradox[J]. Biochim Biophys Acta, 2016, 1865(1): 14-22. DOI: 10.1016/j.bbcan.2015.10.001. [7] Podolskiy DI, Lobanov AV, Kryukov GV, et al. Analysis of cancer genomes reveals basic features of human aging and its role in cancer development[J]. Nat Commun, 2016, 7: 12157. DOI: 10.1038/ncomms12157. [8] LópezOtín C, Galluzzi L, Freije JM, et al. Metabolic control of longevity[J]. Cell, 2016, 166(4): 802-821. DOI: 10.1016/j.cell.2016.07.031. [9] Klutstein M, Nejman D, Greenfield R, et al. DNA methylation in cancer and aging[J]. Cancer Res, 2016, 76(12): 34463450. DOI: 10.1158/0008-5472.CAN-15-3278. [10] Kaur A, Webster MR, Marchbank K, et al. sFRP2 in the aged microenvironment drives melanoma metastasis and therapy resistance[J]. Nature, 2016, 532(7598): 250-254. DOI: 10.1038/nature17392. [11] Lin HK, Chen Z, Wang G, et al. Skp2 targeting suppresses tumorigenesis by Arfp53independent cellular senescence[J]. Nature, 2010, 464(7287): 374-379. DOI: 10.1038/nature08815. [12] Alderton GK. Tumour immunology: give it a rest[J]. Nat Rev Immunol, 2013, 13(3): 156-157. DOI: 10.1038/nri3414. [13] Olshansky SJ. Ageing: measuring our narrow strip of life[J]. Nature, 2016, 538(7624): 175-176. DOI: 10.1038/nature19475. [14] van Deursen JM. The role of senescent cells in ageing[J]. Nature, 2014, 509(7501): 439-446. DOI: 10.1038/nature13193. [15] Chakradeo S, Elmore LW, Gewirtz DA. Is senescence reversible?[J]. Curr Drug Targets, 2016, 17(4): 460-466. [16] GarcíaPrat L, MartínezVicente M, Perdiguero E, et al. Autophagy maintains stemness by preventing senescence[J]. Nature, 2016, 529(7584): 37-42. DOI: 10.1038/nature16187. [17] Serrano M. Unraveling the links between cancer and aging[J]. Carcinogenesis, 2016, 37(2): 107. DOI: 10.1093/carcin/bgv100. [18] Franco A, Kitsis RN, Fleischer JA, et al. Correcting mitochondrial fusion by manipulatingmitofusin conformations[J]. Nature, 2016, 540(7631): 74-79. DOI: 10.1038/nature20156. [19] Cao LL, RiascosBernal DF, Chinnasamy P, et al. Control of mitochondrial function and cell growth by the atypical cadherin Fat1[J]. Nature, 2016, 539(7630): 575-578. DOI: 10.1038/nature20170. [20] Komor AC, Badran AH, Liu DR. CRISPRbased technologies for the manipulation of eukaryotic genomes[J]. Cell, 2017, 168(12): 20-36. DOI: 10.1016/j.cell.2016.10.044. [21] Suzuki K, Tsunekawa Y, HernandezBenitez R, et al. In vivo genome editing via CRISPR/Cas9 mediated homologyindependent targeted integration[J]. Nature, 2016, 540(7631): 144-149. DOI: 10.1038/nature20565. [22] Stahl EC, Brown BN. Cell therapy strategies to combat immunosenescence[J]. Organogenesis, 2015, 11(4): 159-172. DOI: 10.1080/15476278.2015.1120046. [23] Jafri MA, Ansari SA, Alqahtani MH, et al. Roles of telomeres and telomerase in cancer, and advances in telomerasetargeted therapies[J]. Genome Med, 2016, 8(1): 69. DOI: 10.1186/s13073-016-0324-x. [24] Dilley RL, Verma P, Cho NW, et al. Breakinduced telomere synthesis underlies alternative telomere maintenance[J]. Nature, 2016, 539(7627): 54-58. DOI: 10.1038/nature20099. [25] Djukic M, Nau R, Sieber C. The ageing immune system[J]. Dtsch Med Wochenschr, 2014, 139(40): 1987-1990. DOI: 10.1055/s-0034-1370283. [26] Tarazona R, SanchezCorrea B, CasasAvilés I, et al. Immunosenescence: limitations of natural killer cellbased cancer immunotherapy[J]. Cancer Immunol Immunother, 2017, 66(2): 233-245. DOI: 10.1007/s00262-016-1882-x. [27] Cerella C, Grandjenette C, Dicato M, et al. Roles of apoptosis and cellular senescence in cancer and aging[J]. Curr Drug Targets, 2016, 17(4): 405-415. [28] Pedersen JK, Engholm G, Skytthe A, et al. Cancer and aging: epidemiology and methodological challenges[J]. Acta Oncol, 2016, 55 Suppl 1: 7-12. DOI: 10.3109/0284186X.2015.1114670. |
No related articles found! |
Viewed | ||||||
Full text |
|
|||||
Abstract |
|
|||||